In the IVF process, many patients often idealize that "as long as the eggs are retrieved, the embryo can be formed, and as long as the embryo is transferred, the pregnancy can be conceived." ”
However, the reality is not so rosy.
Even if you have a good "seed" (embryo), there is a key link that needs to be overcome in order for it to take root and sprout in the womb and eventually develop into a "big tree" (pregnancy and delivery) - endometrial receptivity.
Endometrial receptivity refers to the ability of the endometrium to accept the embryo. Common diseases such as hydrosalpinx, endometriosis, uterine fibroids, polycystic ovary syndrome, endometritis, and sexual miscarriage have adverse effects on endometrial receptivity.
Since endometrial receptivity is so important for successful conception, can we do something to improve endometrial receptivity and increase our chances of conception?
At the moment, we can only start from the perspective of looking for potential ** and take steps to eliminate them. For example, a patient with hydrosalpinx may undergo laparoscopic surgery** with hydrosalpinx.
We know that hydrosalpinx is caused by chronic inflammation of the fallopian tubes, which may flow backwards into the uterine cavity, where inflammatory factors directly cause damage to the endometrium. It's like a sewage channel that has wreaked havoc on fertile land, making it difficult for even the best land and seeds to germinate. Only when the inflow of sewage is blocked can the seeds take root and sprout.
However, due to the limitations of medical progress, endometrial receptivity is still a research stage, and there is no method that can be widely used in clinical practice and effectively detect endometrial receptivity. This limits the chances of IVF success. We look forward to breakthroughs in this area to improve the success rate of IVF.