Introduction:
The macrophage colony-stimulating factor 2 receptor (CSF2RA) recombinant protein is an important immunomodulatory protein produced by genetic engineering. It plays a key role in the immune system and is important for regulating the development and function of macrophages. In this article, we will introduce the characteristics of CSF2RA recombinant protein and its potential in immunology.
Catalog Number: PA1000-737
Characteristics of CSF2RA Recombinant Protein:
The CSF2RA recombinant protein is a genetically modified protein from humans that is similar to naturally occurring macrophage colony-stimulating factor 2 (CSF2). CSF2 is a cytokine that is widely found in a variety of tissues and cell types, such as macrophages, dendritic cells, and T cells. CSF2RA is a ligand receptor for CSF2 and plays an important role in the development and functional regulation of macrophages.
Application of CSF2RA recombinant protein in immunology**:
Due to its regulatory effect on macrophages, CSF2RA recombinant protein has become a research hotspot in the field of immunology. Studies have shown that CSF2RA recombinant protein can be used to improve the activity of immune cells and promote inflammatory response, thereby enhancing the body's immune defense against infection.
Conclusion: As an important immunomodulatory protein, CSF2RA recombinant protein plays an important role in regulating macrophage development and function. With the in-depth study of its function, the CSF2RA recombinant protein is expected to exert greater potential in immunity and provide people with more effective means.
References: 1 robb l, et al. gm-csf, il-3, and il-5 family of cytokines: regulators of inflammation. immunity, 2018; 50(4): 796–811.
2. edelson bt, et al. batf3-dependent cd11b+ dendritic cells are required for the induction of t(h)17 cells in a mouse model of bacterial infection. nat immunol, 2011; 12(8): 647-54.
3. nair-gupta p, et al. tlr signals induce phagosomal mhc-i delivery from the endosomal recycling compartment to allow cross-presentation. cell, 2014; 158(3): 506-21.