When the liver begins to disease, people are prone to the symptoms of jaundice, but some problems that seem to be jaundice are easily confused with jaundice, and it is necessary to pay attention to distinguish.
Patients with jaundice are often exposed to damp heat and epidemic poison externally, injured by food and drink, and blood stasis or sand and stones in the body are blocked, resulting in obstruction of liver and gallbladder qi, abnormal excretion, and bile overflow. Dampness and blood stasis are the main pathological factors, and the diseased organs mainly involve the spleen, stomach, liver and gallbladder.
It is manifested as yellow staining of the eyes, yellow body and yellow urine. Among them, yellow eyes are the primary symptom, if the body is yellow and the urine is yellow but there is no yellow eyes, it does not belong to jaundice syndrome. It may be accompanied by chills and fever, or loss of appetite, stomach distension, or right upper quadrant or right hypochondrium distension and pain, similar to cold, stomach pain, or hypochondriac pain.
Patients often have a history of unclean or unhealthy diet, exposure to hepatitis patients, or exposure and use of chemicals and drugs, or history of cholecystitis, cholelithiasis, and cirrhosis.
1. Pseudojaundice: mainly due to eating too much carrot, pumpkin, papaya, citrus and other foods, but this group of people does not have sclera jaundice, and serum bilirubin will not rise. Taking some drugs can cause **yellowing, especially the yellow color of the exposed parts of the body is more obvious, but there is no scleral jaanzonic and elevated serum bilirubin.
2. Physiological jaundice: newborns often have this situation, because the neonatal hepatocytes are not yet mature, most newborns have jaundice within 1 week of birth, often reaching a peak in 2-4 days, and then declining after 1 week. After the pathological jaundice is ruled out, physiological neonatal jaundice does not need to be special**, and can be relieved by more sun exposure.
3. Constitutional jaundice: it is a hereditary chronic mild indirect bilirubinemia, which can generally be aggravated after starvation, surgery, fever and exertion. Serological testing showed that the level of indirect bilirubin was high, and the liver morphology was normal on ultrasonography. Generally, it can be recovered after a simple **, no need for **.