Herpes zoster is caused by reactivation of the varicella-zoster virus (VZV). After initial infection, the virus lurks in the ganglia, and when the body's immunity is weakened, the virus is reactivated, leading to the development of shingles.
The pathological process of shingles mainly consists of blisters, papules and pain on the **. The virus affects the ganglia, causing neuralgia and then blisters and pimples on the **. These symptoms usually resolve on their own within a few weeks, but some people may experience pain that lasts for months or longer, which is known as postherpetic neuralgia (PHN).
Pain characteristics: The pain of shingles is usually intense and worsens with movement, touch, or temperature changes. The nature of the pain may include pins and needles, cuts, burning, or electric shocks. The pain of PHN is usually constant and can manifest as a knife, pinprick, tear, or burning pain.
Methods: Methods for shingles include antivirals, pain relievers, topical, and immune. Antiviral drugs such as acyclovir or valacyclovir can reduce the amount of virus and reduce symptoms. Pain relievers such as over-the-counter medications such as acetaminophen or prescription medications such as ***madol, gabapentin, etc., can help relieve pain. Topical** includes dryness, anti-inflammatory, and protection against secondary infection. Immunization** includes the use of immune boosters such as interferon or interleukins to boost the immune system's response. For PHN, antiepileptic medications, antidepressants, or neuroleptics may be required**.
Please note that the above content is for informational purposes only and does not constitute professional medical advice. If necessary, seek medical attention promptly and follow your doctor's instructions.