Step 1: HPV infection.
Low-risk HPV infection: 6, 11, 40, 42, 43, 54, 61, 70, 72, 81, CP6108.
About 90% of ** warts are caused by HPV types 6 and 11.
High-risk HPV infection:
Female** infected with high-risk HPV can easily lead to cervical precancerous lesions and cervical cancer, especially 16,18
Lesions associated with low-risk HPV infection are predominantly CINI, and most may be reversed;
CIN associated with high-risk HPV infection tends to progress.
Step 2: CINI- (low-grade squamous intraepithelial lesion, LSIL)
It is a condition associated with HPV infection, and the lesions at this stage have a great possibility of reversal, and most of them will disappear on their own.
Both CIN2 and CIN3 belong to (high-grade squamous epithelial lesions, HISL).
Step 3: CINII
Intervention is required**, and physical is an option for grade 2 disease.
Step 4: Ciniil
Excision is performed by leep circumcision or cold knife conization**.
Step 5: Cervical cancer.
If cervical cancer is found at an early stage, it can also be obtained by surgery**.
The key is to detect early.
Pathological changes in cervical cancer:
Cervical epithelium atypical hyperplasia (mild-moderate-severe) - carcinoma in situ - early invasive cancer - invasive cancer CIN is a precancerous lesion of the cervix, which is an intermediate link in the progression of cervical epithelium to invasive cancer.
There are three tendencies in the CIN development process:
Natural regression or reversal;
Constant;Progression to higher-grade CIN and even invasive cancer.
Correction 1: It is not necessary for CIN to develop into invasive cancer, and not all invasive cancers are transformed from Ciniil (carcinoma in situ), and some are directly transformed from CINI or CINI.
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